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Clifford L. Slayman

Professor of Cellular & Molecular Physiology

  • A.B. 1958 Kenyon College

  • Ph.D. 1963 Rockefeller University

Contact Info: Courier Address:

Phone:  (203) 785-4478

Lab:  (203) 785-2900

Fax:  (203) 785-4951

e-mail:  clifford.slayman@yale.edu

333 Cedar Street,

SHM B128/130

New Haven, CT  06510

 

The molecular machines which mediate small-molecule traffic across biological membranes almost all either create or dissipate electric fields within those membranes, and the resulting changes of trans­membrane voltage or current can be used not only to monitor the underlying traffic, but also to char­acterize changes in the molecular structure of the machines themselves. Micororganisms offer a vast resource for such measurements and analysis: because of their abundance and variety, and because many of them maintain very large transmembrane voltages—in the range of 150 to 300 mV (c.f., ~80 mV in animal cells). Such voltages are used to drive the accumulation of nutrients, the expulsion of wastes and toxins, and in some cases the release of useful secretory products.

Our laboratory is presently using electrophysiological techniques as primary assays of ion-transport processes in three different microorganisms: Neurospora (bread mold), Candida (a facultative human pathogen), and Saccharomyces (baker's yeast); and it is using site-directed mutagenesis to manipulate the structures of a variety of membrane proteins: potassium transporters (TRK & HAK classes), proton pumps (PMA) and potassium channels (TOK).

We are also exploring the molecular mechanisms of action of newly emerging elements of innate immunity, viz ., so-called antimicrobial peptides (AMPs), which serve dual roles in directly attacking invading bacteria, fungi, and protists, and in signaling to adaptive immunity. Many of these agents (more than a thousand are now known) also have multiple modes of action within target micro­organ­isms, which makes them relatively resistant to microbial adaptation. Several classes of these AMPs, and synthetic analogues of them, have great promise for clinical application.

Figure caption:

Structural model of an assembled tetramer of yeast TRK protein, SpTrk1p (from Schizosac­char­o­myces ). A) perspective of the membrane components of the whole tetramer. White (back­ground) = stick-figure representation of six transmembrane helices in each of the four monomers. Magenta = clustered P-loops forming the functional K + pathways. Red & Green = clustering of the remaining 2 x 4 transmembrane helices, to form a central pore. B,D) Ribbon diagrams of the eight clustered helices, designated M1d (inner) and M2d (outer), viewed (B) down the axis of the central pore from the intra­cellular surface of the yeast plasma membrane; and viewed (D) in axial (longitudinal) section from within the plane of the membrane. C,E) Space-filling diagrams (H atoms omitted) corresponding to B,D . Green = neutral amino acids, Red = basic amino acids, Blue = acidic amino acids. Blue balls indicate the putative pathway for chloride transit. Coordinates from H.R. Guy & S.R. Durell ( Biophys. J. 77 : 789, 1999). Drawing by A. Rivetta ( Biophys. J . 89 : 2412, 2005).

 

Selected publications:

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Bertl, A., Bihler, H. Kettner, C., & Slayman, C.L. , 1998. Electrophysiology in the eukaryotic model cell, Sac­charomy­ces cerevisiae . Pflügers Archiv Eur. J. Physiol . 436 : 999-1013. PMID: 9799419

Kettner, C., Bertl, A., Obermeyer, G., Slayman, C.L., & Bihler, H., 2003. Electrophysiological analysis of the yeast V-type proton pump: Variable coupling ratio and proton shunt. Biophys. J . 85 : 3730-3738. PMID: 14645064

Kuroda, T., Bihler, H., Bashi, E., Slayman, C.L. , & Rivetta, A., 2004. Chloride channel function in the yeast TRK-potassium transporters. J. Membr. Biol . 198 :177-192. PMID: 15216418

Baev, D., Rivetta, A., Vylkova, S., Sun, J.N., Zeng, G.-F., Slayman, C.L. , & Edgerton, M., 2004. The TRK1 potassium transporter is the critical effector for killing of Candida albicans by the cationic protein, Histatin 5. J. Biol. Chem . 279 :55060-55072. PMID: 15485849

Roller, A., Natura, G., Bihler, H., Slayman, C.L. , Eing, C., & Bertl, A., 2005. In the yeast potassium channel, Tok1p, the external ring of aspartate residues modulates both gating and conductance. Pflügers Arch.— Europ.J.Physiol . 451 :362-370. PMID: 16133265

Rivetta, A., Slayman, C.L. , & Kuroda, T., 2005. Quantitative modeling of chloride conductance in yeast TRK potassium transporters. Biophys. J . 89 :2412-2426. PMID: 16040756

Roller, A., Natura, G., Bihler, H., Slayman, C.L., & Bertl, A., 2008. Functional consequences of leucine and tyrosine mutations in the dual pore motifs of the yeast K + channel, Tok1p. Pflügers Arch.— Europ.J.Physiol . 456 ::883-896 . PMID: 18421473

 

clifford.slayman@yale.edu

 
 
Department of
Cellular & Molecular
Physiology

Yale University
School of Medicine
333 Cedar Street,
Room B-147
P.O. Box 208026
New Haven, CT
06520-8026

(203) 785-2989 Tel.
(203) 785-4951 Fax
   
       
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Last modified:  October 26, 2009  (cla)